Aspiration Pneumonia and Lung Abscess
Aspiration pneumonia may be due to inhalation of gastric juice or oropharyngeal contents, foreign body aspiration, and the spontaneous intrabron-chial drainage of purulent material from a diseased lung to the contralateral healthy lung. Impairment of the airway’s protective mechanisms, especially cough and glottic closure, is the major predisposing factor and is most often seen in patients with neurological disorders or acute loss of consciousness, especially in the presence of poor dentition. Pulmonary injury includes chemical pneumonitis, necrotizing pneumonia, lung abscess, and empyema. Anaerobic bacteria are the major pathogens in community-acquired aspiration pneumonia, while aerobic organisms such as S. aureus, S. pyogenes, and various GNB are less commonly responsible but predominate in hospitalized patients. Onset of disease is insidious, beginning as an acute pneumonic process with the development of an abscess or empyema after one to two weeks. Dependent areas of the lung are particularly involved. The sputum and discharges are usually putrid and foul-smelling, but this is not invariable. Diagnosis of bacterial etiology should not be based on sputum examination because of the frequency of contamination by upper airway organisms. If a specific bacteriological diagnosis is desired, invasive techniques such as transtracheal aspiration, transthoracic aspiration, or bronchoscopy are required. Since S. aureus and GNB are so easily recovered from mixed culture samples, their absence in a purulent specimen usually excludes their presence in the lower respiratory tract. The converse cannot be said, however, and their growth should not be taken as an indication of their involvement. Treatment is usually empirical and consists of penicillin G, which is effective even against organisms such as B. /ragilis that display in vitro resistance. Clindamycin is a reasonable alternative agent. Surgical resection of the abscess cavity or decortication of the pleural peel is rarely required.
- Endocrine Systems
- GROSS ANATOMY
- Clinical Course, Pathogenesis, and Anatomy of Acute Tubular Necrosis
- PRE-EXCITATIOIi SYNDROMES
- Factors Involved in the Choice of Type of Dialysis
- C. MALABSORPTION
- Pyuria
- Ovarian Cancer
- EMBOLIC DISEASE
- OTHER ESOPHAGEAL DISORDERS
- Urinary Tract Infection
- Portal Hypertension
- Pathogenic Mechanisms - Mechanism of Injury
- PHYSIOLOGY OF THE SYSTEMIC CIRCULATION
- CARDIAC TRAUMA
- CARCINOMA OF THE PANCREAS - Diagnosis
- Ascites
- Phenytoin
- PERIPHERAL VENOUS DISEASE
- DIAGNOSTIC TECHNIQUES AND THEIR INDICATIONS - IMAGING PROCEDURES
- ARTERIAL TRAUMA
- Renal Biopsy
- HEART BLOCK
- PLEURAL EFFUSIONS
- Liddle’s Syndrome
- PROGNOSIS
- PERIPHERAL ANEURYSMS AMD FISTULAE
- CARDIAC TUMORS
- AORTIC ARTERITIS
- ACUTE PANCREATITIS
- CLINICAL APPROACH TO LIVER DISEASE
- THE APPROACH TO THE PATIENT WITH GASTROINTESTINAL HEMORRHAGE
- DRUGS
- CLINICAL CLASSIFICATION OF JAUNDICE
- SMOKE INHALATION