VASCULAR DISEASE OF THE LIVER
Portal vein thrombosis, hepatic vein thrombosis (Budd-Chiari syndrome), and veno-occlusive disease are uncommon disorders of hepatic vasculature that usually present with portal hypertension with or without associated liver dysfunction.
Portal vein thrombosis may develop after abdominal trauma, umbilical vein infection, sepsis, or pancreatitis or in association with cirrhosis or hypercoagulable states; in most cases, however, and particularly in children, the cause is unknown. The disease presents with the manifestations of portal hypertension (see Chapter 46); however, liver histology is usually normal. The diagnosis is established by angiography. Surgical management is difficult owing to the absence of suitable patent vessels for portal-systemic shunting.
The BuddChiari syndrome is associated with abdominal trauma, use of oral contraceptives, polycythemia vera, paroxysmal nocturnal hemoglobinuria, other hypercoagulable states, and congenital webs of the vena cava. Illness may be acute or chronic with abdominal pain, hepatomegaly, ascites, and portal hypertension as prominent features. The diagnosis is usually suspected when centrilobular necrosis is seen on liver biopsy and is established angiographically by inability to catheterize the hepatic veins. Although elevation of serum bilirubin and transaminases is often mild, liver function is often poor and mortality rates of 40 to 90 per cent are reported. Anticoagulants have not proven useful; however, recent studies have suggested that the side-to-side portacaval shunts, performed to relieve hepatic congestion, may improve survival.
Veno-occlusive disease or nonthrombotic occlusion of hepatic venules is a smallvessel variant of the Budd-Chiari syndrome. Veno-occlusive disease develops in man and animals exposed to native medicinal teas containing pyr-rolozidine alkaloids from Senecio and Crotalia genera of plants. Cases have also been reported in association with the use of certain chemothera-peutic agents and with bone marrow transplantation. The occluded venules can be seen on liver biopsy, and a distinctive abnormal vascular pattern may be seen when contrast is injected into the hepatic vein. Patients with alcoholic liver disease also frequently exhibit some degree of hepatic venule sclerosis or occlusion. No specific treatment is available. Some patients appear to recover spontaneously, whereas others require therapy for the complications of portal hypertension.
- Alterations in Glomerular Hemodynamics, Parathyroid Hormone Metabolism, and Systemic Arterial Blood Pressure
- RISK FACTORS
- The Fanconi Syndrome
- CHROMC BROriCMITIS
- PLEURAL EFFUSIONS
- Incidence
- SPECIFIC CAUSES OF CIRRHOSIS
- PATHOLOGY
- ATRIAL RHYTHM DISTURBANCES
- MOTOR DISORDERS OF THE ESOPHAGUS
- Lidocaine
- Diagnosis
- PATHOPHYSIOLOGY
- PERIPHERAL ANEURYSMS AMD FISTULAE
- BRORICHODILATORS
- PATHOGENESIS OF RESPIRATORY TRACT INFECTION
- Studies of Pancreatic Structure and Function
- SOLITARY PULMONARY NODULE
- Classification or Glomerular Diseases
- Polycystic Kidney Disease (PKD)
- RENAL PHARMACOLOGY
- CLINICAL PRESENTATION
- Tocainide
- Factors Involved in the Choice of Type of Dialysis
- Progressive Crescentic Glomerulonephritis
- ANGINA PECTORIS
- SPECIFIC ARRHYTHMIAS - sinus nodal rhythm disturbances
- Diagnosis
- Other Clearly Extrinsic Causes of Diffuse Infiltrative Lung Disease
- CARDIOVASCULAR PHYSIOLOGY DURING PREGNANCY - ELECTROPHYSIOLOGY
- Pulmonary Hemorrhagic Disorders
- Pathology
- APPROACH TO THE PATIENT WJTH SUSPECTED MALDIGESTION AND/OR MALABSORPTION
- CLINICAL TESTS OF DIGESTION AND ABSORPTION
- Minimal Change Nephropathy