Liver Failure
The derangement of hepatocellular function that occurs in cirrhosis manifests as impaired hepatic protein synthesis (hypoalbuminemia, impaired synthesis of coagulation factors) and failure of normal hepatic detoxification processes. The latter accounts for the development of signs such as spider angiomata and contributes to the development of hepatic encephalopathy, as well as to poorly understood hemodynamic and hormonal disorders that foster the development of ascites and the hepatorenal syndrome.
- TREATMENT
- Pyuria
- Important NEPHROTOXIRIS
- Etiology and Pathogenesis
- PERIPHERAL VENOUS DISEASE
- Portal Hypertension
- CLINICAL MANIFESTATIONS
- Familial Polyposis of the Colon
- Renal Biopsy and Other Diagnostic Tests
- RADIOGRAPHIC AND ENDOSCOPIC PROCEDURES IN GASTROENTEROLOGY
- Hematopoietic System
- Ovarian Cancer
- THE APPROACH TO THE PATIENT WITH GASTROINTESTINAL HEMORRHAGE
- Endoscopic “Retrograde” Cholangiopancreatography (ERCP)
- CLINICAL SYMPTOMS OF ESOPHAGEAL DISEASE
- BROliCHIECTASIS
- VENTILATION
- PERIPHERAL ANEURYSMS AMD FISTULAE
- RHEUMATIC FEVER
- HEART BLOCK
- CHROMIC PANCREATITIS
- Renal Glycosuria
- Vitamin Dresistant Rickets
- APPROACH TO THE DIAGNOSIS OF JAUNDICE
- Initial Assessment
- NORMAL ABSORPTION
- Radionuclide Imaging
- DISORDERS ASSOCIATED WITH MALABSORPTION
- SPECIFIC PATHOGENIC ORGANISMS
- GRANULOMATOUS LIVER DISEASE
- SPECIFIC CLINICAL DISORDERS
- PNEUMOTHORAX
- Aminoaciduria
- INVASIVE DIAGNOSTIC TECHNIQUES
- Blood Chemistries