GENERAL PRINCIPLES OF CARDIAC SURGERY
Surgical results are excellent in simple abnormalities such as uncomplicated atrial septal defects, but the mortality and morbidity are higher for repair of more complex lesions such as te-trology of Fallot. Results also depend oh whether the surgery is corrective or palliative; i.e., lesions that are incompletely corrected at the time of surgery inherently have less favorable outcome. Overall ventricular function is one of the most important determinations of cardiac surgical risk.
The prognosis for postoperative recovery of left ventricular function differs depending on the underlying disease. For example, left ventricular function may not improve postoperatively in a patient with a large myocardial scar from previous infarction, whereas the patient with left ventricular dysfunction secondary to aortic stenosis is likely to show improvement in left ventricular function upon correction of the aortic stenosis.
The general medical status of the patient may affect cardiac surgical results. For example, abnormal pulmonary function is a common cause of complications after cardiac surgery. In addition to pre-existing primary lung diseases such as chronic obstructive pulmonary disease, pulmonary hypertension secondary to heart disease may increase the risk of surgery. Severe irreversible pulmonary hypertension from left-to-right shunts (for example, ventricular septal defect) is a contraindication to surgery, but pulmonary hypertension and increased pulmonary vascular resistance secondary to mitral stenosis usually resolves after surgical correction. The pre-existence of chronic renal failure increases the risk of acute renal failure in the postoperative period. Even mild hepatic disease may be exacerbated after cardiac surgery, increasing the risk of acute hepatic failure. Preexisting neurological defects may be exacerbated by cardiopulmonary bypass but usually eventually return to baseline. In patients with serious carotid artery obstruction, care must be taken to maintain flow during cardiopulmonary bypass to minimize the risk of neurological injury.
All noncardiac medical conditions should be investigated and treated before cardiac surgery. Most medications required for optimal medical management should be administered up until the evening before or day of surgery, including beta blockers and antihypertensive medication. Continuous monitoring of the electrocardiogram, arterial pressure, central venous pressure, and pulmonary arterial and wedge pressures is usually initiated prior to induction of anesthesia to obtain baseline values. An intra-aortic balloon counterpulsation device may be inserted preoperatively in selected high-risk patients.
Complications of cardiopulmonary bypass involve those related to the cannulation and the technical institution of bypass and those related to the secondary effects of extracorporeal circulation on various organ systems. An example of the first category is a dissection of the aorta while introducing the aortic cannula. Pulmonary, renal, or neurological dysfunction is common following cardiopulmonary bypass, especially if disease is pre-existent.
Convalescence after cardiac surgery depends on the extent of pre-existing cardiac and noncardiac disease, the direct trauma of the operation, and the response to cardiopulmonary bypass. The thermodirution cardiac output should be periodically monitored in the early postoperative hours. The adequacy of arterial pressure, peripheral pulses, skin temperature, urinary output, arterial blood gases, and mixed venous oxygenation is periodically assessed. Arterial pressure may be apoor indicator of adequate output after surgery because peripheral arterial resistance may be markedly elevated. If cardiac output is insufficient, the patient may require fluids, afterload reduction, inotropic drugs and/or improvement in oxygenation. Measurement of left ventricular filling pressure is important to assess adequacy of fluid volume. If bradycardia is present, pacing via surgically implanted temporary wires (preferably atrial or AV sequential pacing) may optimize cardiac output. If pharmacological manipulation does not improve cardiac output, intra-aortic balloon counterpulsation may be required and may be of more value than inotropic agents if ischemia or arrhythmias are a particular problem. Tachyarrhythmias must be treated vigorously. Exacerbating factors such as hypoxia, anemia, or electrolyte abnormalities should be corrected. The pacing wires can be used to terminate atrial tachyarrhythmias such as atrial flutter! Complex ventricular arrhythmias are treated with lidocaine as the drug of first choice. Atrial tachyarrhythmias may require treatment with digoxin, beta blockers, or in some cases membrane-active agents such as procainamide.
Ventilation with positive end-expiratory pressure should be employed but used cautiously in patients with chronic obstructive pulmonary disease. Patients are usually extubated within four to eight hours after surgery but sometimes not until the next morning.
If bleeding occurs postoperatively, examination of hematological parameters such as fibrinogen, platelets, and coagulation times should be obtained. Blood and blood products should be administered as indicated. Drainage from pleural and pericardial tubes should be monitored, and if excessive the patient may need to be returned to surgery for exploration. Cardiac tamponade is an occasional complication requiring early reoperation.
- Other Glomerulonephritides
- INFECTIVE ENDOCARDITIS
- CLINICAL MANIFESTATIONS OF ENDSTAGE RENAL DISEASE
- Treatment
- PHYSIOLOGY OF THE SYSTEMIC CIRCULATION
- ACUTE PANCREATITIS
- Clinical Manifestations
- Mesangioproliferative Glomerulonephritis
- GENERAL SURGERY IN THE PATIENT WITH HEART DISEASE
- Gardner's Syndrome
- Beta Blockers
- CARCINOMA OF THE PANCREAS - Definition
- Urinary Tract Obstruction
- ACUTE RENAL INSUFFICIENCY
- PHYSIOLOGY OF THE CORONARY CIRCULATION
- Gastrointestinal Tract
- EMBOLIC DISEASE
- PEPTIC ULCER DISEASE OF THE STOMACH AND DUODENUM
- NONPENETRATING TRAUMA
- RENAL PHARMACOLOGY
- PHYSIOLOGICAL EFFECTS OF PULMONARY HYPERTENSION ON CARDIAC FUNCTION
- Pathogenic Mechanisms - Mechanism of Injury
- Liddle’s Syndrome
- SMOKING CESSATION
- Hepatic Encephalopathy
- Indirect
- SCREENING TESTS OF HEPATOBILIARY DISEASE
- SPECIFIC ARRHYTHMIAS - sinus nodal rhythm disturbances
- Anatomical Imaging of the Urinary
- CLINICAL MANIFESTATIONS OF MALABSORPTION
- Endocrine and Other Considerations
- SPECIFIC PATHOGENIC ORGANISMS
- NONRESPIRATORY FUNCTIONS OF THE LUNG
- INVASIVE DIAGNOSTIC TECHNIQUES
- Classification or Glomerular Diseases