Liddle’s Syndrome
As in Bartter’s syndrome, hypokalemia, hyper-kaliuria, and hypochloremic metabolic alkalosis characterize this rare disorder. However, in sharp contrast to Bartter’s syndrome, aldosterone levels are normal, renal avidity for sodium is increased, and patients are hypertensive in Liddle’s syndrome. A primary increase in distal nephron sodium absorption, which can be blocked by the diuretic triamterene but not by the aldosterone antagonist spironolactone, appears to be the cause of the unusual clinical syndrome.
- LIMITATION OF MFARCT SIZE
- OXYGEN THERAPY AND MECHANICAL VENTILATION
- SYNCOPE
- NONOBSTRUCTIVE CAUSES OF ISCHEMIC HEART DISEASE
- Plain Radiographs and Barium Contrast Studies
- Pyuria
- ENDOSCOPIC PROCEDURES
- SPECIFIC PATHOGENIC ORGANISMS
- Proteinuria
- History and Physical Examination
- Renal Artery Stenosis
- CONTROL OF BREATHING IN DISEASE STATES
- LABORATORY TESTS IN LIVER DISEASE
- Incidence
- Mesangioproliferative Glomerulonephritis
- ATHEROSCLEROSIS
- NONPHARMACOLOGICAL THERAPY OF TACHYARRHYTHMIAS
- RENAL PARENCHYMAL
- MEDICAL MANAGEMENT OF ANGINA
- Resuscitation
- CAUSES OF PULMONARY HYPERTENSION
- Urinary Tract Obstruction
- CARDIOMYOPATHY
- Hepatocellular Carcinoma
- Renal Biopsy
- PROSTHETIC VALVES
- GRANULOMATOUS LIVER DISEASE
- ORIGIN OF ABDOMINAL PAIN
- Diagnosis
- Lower GI Bleeding
- Direct (Toxic Nephropathy)
- Beta Blockers
- Renal Artery Occlusion
- THE ZOLLINGER-ELLISON SYNDROME
- Community Acquired Pneumonia