MANAGEMENT OF CARDIAC ARRHYTHMIAS
Before initiating antiarrhythmic therapy, one must determine whether the arrhythmia should be treated. Any arrhythmia that causes symptomatic hypotension or sudden death should be suppressed. However, the situation in which the arrhythmia occurs dictates whether chronic, long-term therapy is necessary. For example, an episode of ventricular fibrillation in a patient at the onset of an acute myocardial infarction does not necessarily require long-term drug therapy because of the low likelihood of recurrence.
However, ventricular fibrillation in a patient without an acute myocardial infarction carries a high risk of recurrence. Some patients may have arrhythmias that, while not life-threatening, produce disabling symptoms of dizziness or palpitations and require therapy. Rhythms that are tolerated well in patients with structurally normal hearts (for example, paroxysms of supraventricular tachycardia) may not be tolerated in patients with diseased hearts (for example, ischemic heart disease or mitral stenosis) and may require therapy. The decision to treat a patient with an asymptomatic tachyarrhythmia is more difficult. Certain arrhythmias, such as short episodes of asymptomatic nonsustained ventricular tachycardia, are in themselves harmless but may be forerunners of more serious sustained ventricular tachyarrhythmias. The decision to treat is complicated by the side effects, occasionally life-threatening, of antiarrhythmic drugs, such as exacerbation of ventricular arrhythmias in 5 to 15 per cent of cases. Even though patients with premature ventricular complexes and complex ventricular ectopy after myocardial infarction are at increased risk of subsequent sudden death, it is not clear that antiarrhythmic treatment reduces the increased mortality.
Before beginning chronic antiarrhythmic therapy, factors contributing to the occurrence of the arrhythmia should be considered. These include digitalis excess, hypokalemia, Hypomagnesemia, hypoxia, thyrotoxicosis, and other severe metabolic derangements. Congestive heart failure, anemia, or infection should be corrected. Smoking, excessive alcohol intake, caffeine- or theophyl-line-containing beverages or foods, fatigue, emotional upset, and some over-the-counter drugs (for example, nasal decongestants) may exacerbate arrhythmias.
- MISCELLANEOUS AORTIC DISEASE
- Nephritic Glomerulopathies
- APPROACH TO THE PATIENT WJTH SUSPECTED MALDIGESTION AND/OR MALABSORPTION
- Diagnosis
- Diagnosis
- DC CARDIOVERSION AND DEFIBRILLATION
- PATHOPHYSIOLOGY
- The Use of Diuretics
- ACUTE PANCREATITIS
- Other Glomerulonephritides
- Comprehensive Health-care Program for Children in Foster Care
- PATHOPHYSIOLOGY OF ISCHEMIC HEART DISEASE
- Lidocaine
- Other Cystic Diseases
- Gardner's Syndrome
- ADAPTATION TO NEPHRON LOSS
- Amiodarone
- Indications for Dialysis and Adequacy of Dialysis
- Pathogenic Mechanisms
- Upper GI Bleeding
- ACUTE MYOCARDIAL INFARCTION
- Metabolism of Drugs in Patients with Renal Insufficiency
- CARDIOMYOPATHY
- ETIOLOGY OF GASTROINTESTINAL BLEEDING
- Aspiration Pneumonia and Lung Abscess
- GRANULOMATOUS LIVER DISEASE
- Verapamil
- PERIPHERAL ANEURYSMS AMD FISTULAE
- Visualization of the Biliary Tree
- Sigmoidoscopy and Colonoscopy
- NONOBSTRUCTIVE CAUSES OF ISCHEMIC HEART DISEASE
- PLEURAL DISEASE
- APPROACH TO THE DIAGNOSIS OF JAUNDICE
- CARCINOMA OF THE COLON
- CYSTIC FIBROSIS