NONPULMONARY FACTORS
Abnormalities other than alterations in lung function may influence the Pao2 through their effect on the mixed venous Po2(Py02). The Pv02 is decreased when cardiac output is inappropriately low, when 02 consumption (Vo2) is increased (as with exercise or fever), or when the hemoglobin concentration or 02 saturation is low. For any lung unit, the resultant end-capillary Po2 is influenced by the Pvo2, although the magnitude of this effect on the arterial 02 content will be greatest in lungs with Va/Q inequality or shunt (Fig. 17-9). The importance of this phenomenon is the recognition that a fall in Pao2 in a patient with lung disease may be due to one of these nonpulmonary factors rather than a deterioration in lung function, thus requiring a very different intervention.
- Anatomical Imaging of the Urinary
- GAS TRANSFER
- Etiology and Pathogenesis
- Mesangioproliferative Glomerulonephritis
- Pulmonary Hemorrhagic Disorders
- CLINICAL FEATURES OF PULMONARY HYPERTENSION
- NORMAL INTESTINAL PHYSIOLOGY
- Incidence
- LIVER BIOPSY
- TREATMENT
- MYOCARDIAL METABOLISM
- CLINICAL PRESENTATION
- Renal Artery Occlusion
- PERICARDIAL DISEASES - ACUTE PERICARDITIS
- Determination of Kidney Anatomy and Renal Blood Flow
- Diagnosis
- Procainamide
- Bartter’s Syndrome
- Lower GI Bleeding
- Renal Glycosuria
- HEART DISEASE AND PREGNANCY
- THE AIRWAY STRUCTURE
- Pathology
- MECHANISMS OF ARRHYTHMOGENESIS
- MULTISYSTEM DISEASE WITH RENAL INVOLVEMENT
- Amyloidosis
- DRUG-ASSOCIATED RENAL INJURY
- Incidence
- CAUSES OF PULMONARY HYPERTENSION
- Mixed Glomerulopathies
- Diet
- RENAL PARENCHYMAL
- CLASSIFICATION AND PATHOPHYSIOLOGY
- CARDIOVASCULAR PHYSIOLOGY DURING PREGNANCY - ELECTROPHYSIOLOGY
- PERIPHERAL VENOUS DISEASE