Liver Failure
The derangement of hepatocellular function that occurs in cirrhosis manifests as impaired hepatic protein synthesis (hypoalbuminemia, impaired synthesis of coagulation factors) and failure of normal hepatic detoxification processes. The latter accounts for the development of signs such as spider angiomata and contributes to the development of hepatic encephalopathy, as well as to poorly understood hemodynamic and hormonal disorders that foster the development of ascites and the hepatorenal syndrome.
- Clinical Course, Pathogenesis, and Anatomy of Acute Tubular Necrosis
- THE AIRWAY STRUCTURE
- Endocrine and Other Considerations
- Clinical Presentation
- MEDICAL MANAGEMENT OF ANGINA
- PATHOPHYSIOLOGY OF ISCHEMIC HEART DISEASE
- Elimination of Waste Products of Metabolism and Drugs
- CARCINOMA OF THE PANCREAS - Clinical Manifestations
- Nephrogenic Diabetes Insipidus (NDI)
- Sigmoidoscopy and Colonoscopy
- THE COMMON CLINICAL MANIFESTATIONS OF GASTROINTESTINAL DISEASE
- SMOKE INHALATION
- MICROSCOPIC ANATOMY
- CLINICAL SYMPTOMS OF ESOPHAGEAL DISEASE
- GLOMERULAR DISEASE
- Ultrasound and Computed Tomography
- Urinary Tract Obstruction
- Renal Venous Occlusion
- Gastrointestinal Tract
- Acid-Base Abnormalities
- Treatment and Prognosis
- Resuscitation
- Visceral Angiography
- Initial Assessment
- ACUTE RENAL INSUFFICIENCY
- AORTIC DISEASE - AORTIC ANEURYSMS
- APPROACH TO THE PATIENT WITH SUSPECTED OR CONFIRMED ARRHYTHMIAS
- Improving Case Management
- HEART BLOCK
- Systemic Lupus Erythematosus (SLE)
- CLINICAL PRESENTATION
- CYSTIC FIBROSIS
- PERICARDIAL EFFUSIOH
- Diet
- Liver Failure