POSTCAPILLARY PULMONARY HYPERTENSION
This results from the passive effects of an elevated left atrial pressure due to mitral valve disease or prolonged left ventricular failure. If it persists, pulmonary artery pressure gradually increases above that explained by passive transmission of the elevated outflow pressure. Initially this increase is due to reflex vasoconstriction, but eventually vascular remodeling occurs. This additional contribution to pulmonary hypertension can be quantified by the increasing gradient between the pulmonary diastolic pressure and the left atrial pressure.
- PULMONARY HEART DISEASE
- Mechanism of Proteinuria
- NONMEDICAL MANAGEMENT OF ANGINA PECTORIS
- Indirect
- Initial Assessment
- Proliferative Glomerulonephritis
- ATHEROSCLEROSIS
- HEMODIALYSIS AND HEMOPERFUSION IN THE TREATMENT OF DRUG OVERDOSES
- Nephrosclerosis
- Ascites
- Hepatic Diseases
- ENDOSCOPIC PROCEDURES
- OXYGEN
- Esophagogastroduodenoscopy
- EMBOLIC DISEASE
- Blood Chemistries
- Pulmonary Infiltrates with Eosinophilia PIE
- Clinical Manifestations
- Gardner's Syndrome
- INVASIVE DIAGNOSTIC TECHNIQUES
- OTHER ESOPHAGEAL DISORDERS
- CLASSIFICATION AND PATHOPHYSIOLOGY
- Urinary Tract Infection
- Nephrogenic Diabetes Insipidus (NDI)
- CARCINOMA OF THE COLON
- CHRONIC RENAL FAILURE
- Plain Radiographs and Barium Contrast Studies
- PHYSICAL EXAMINATION
- Systemic Vasculitides
- MEDIASTINAL DISEASE
- VASCULAR DISEASE OF THE LIVER
- CLINICAL MANIFESTATIONS
- Endoscopic “Retrograde” Cholangiopancreatography (ERCP)
- Nephritic Glomerulopathies
- TRAMSPLATTTATION