PRINCIPLES OF CARDIOPULMONARY RESUSCITATION
Cardiopulmonary resuscitation consists of basic and advanced life support. Upon evaluating a patient with suspected cardiac arrest, one should first quickly establish that the patient is truly unresponsive and not breathing. If a pulse is not present, a precordial thump to the mid-sternum may be tried. Subsequently, the “ABC’s” of basic life support should be observed: Airway, Breathing, and Circulation. The mouth and pharynx should be examined to assure that no obstruction is present. The tongue should be removed from the posterior pharynx by tilting the head backward and hyperextending the neck. This maneuver can sometimes cause resumption of spontaneous respiration. If no breathing is noted, mouth-to-mouth or mouth-to-nose breathing should be initiated in four quick breaths. Time is often wasted trying to intubate a patient when adequate ventilation could be accomplished immediately via mouth or mask ventilation. One should check to see that the chest rises with each ventilation. If a carotid pulse is not present after the initial ventilations, external cardiac compression over the lower half of the sternum (not over the xiphoid process) should be initiated. The sternum should be depressed 3 to 5 cm, with the patient lying on a hard surface. Compressions should be approximately 60 per minute, with a ratio of 5 compressions to 1 ventilation if two rescuers are present. A single rescuer must give 15 chest compressions alternating with two ventilations every 15 seconds.
Advanced life support should be initiated while basic life support continues. Defibrillation should be applied if indicated as soon as possible and is the single most definitive treatment available for most cardiac arrests. Oxygen should be administered and an adequate intravenous access should be established. If circulation has not been restored quickly, sodium bicarbonate 1 mEq/kg IV is given to treat metabolic acidosis and is repeated after 10 minutes; further administration of sodium bicarbonate should be guided by blood gas and pH measurements once effective circulation is restored. Epinephrine (5 to 10 ml of a 1:10,000 solution administered via an intravenous, intracardiac, or endotracheal route every 5 minutes as needed) is useful in treating asystole and also in aiding defibrillation of fine (low-amplitude) ventricular fibrillation. Atropine (boluses of 0.5 mg IV at 5-minute intervals to a total dose of approximately 2 to 4 mg) can be administered for profound bradycardia. Isoproterenol given as a constant infusion (2 to 20 |i,g/min) and titrated according to response may be used to treat brady-arrhythmias if atropine is ineffective. Emergency cardiac pacing may be attempted for bradyar-rhythmias if atropine and isoproterenol are unsuccessful.
Lidocaine, procainamide, or bretylium tosylate can be administered to help terminate ventricular tachyarrhythmias and prevent their recurrence. Intravenous furosemide and/or morphine may be used to relieve pulmonary edema. Calcium chloride (2.5 to 5 ml of a 10 per cent solution repeated if necessary in 10 minutes) is given to increase myocardial contractility, especially if electromechanical dissociation is present. Calcium should be used with caution in a patient with known digitalis excess. Calcium chloride will precipitate if given in the same intravenous line with sodium bicarbonate.
Electromechanical dissociation refers to the presence of cardiac electrical activity without appropriate mechanical activity. It may be caused by decreased filling of the heart (e.g., hypovolemia, cardiac tamponade, pulmonary embolus) or severe myocardial pump depression that may respond to calcium. Emergency pericardiocentesis may be attempted if cardiac tamponade is suspected.
The widespread application of cardiopulmonary resuscitation via education of the public and extensive emergency care systems in many cities has increased both the number of cardiac arrest victims who reach the hospital and the number who survive to be discharged. Survival critically depends on the time from arrest to the initiation of resuscitation and is best if basic life support can be initiated within 3 to 4 minutes and more definitive therapy (i.e., defibrillation) shortly thereafter.
- VARIATiT ANGINA
- PERICARDIAL DISEASES - ACUTE PERICARDITIS
- OBSTRUCTIVE LUNG DISEASE
- Acid-Base Abnormalities
- Etiology and Pathogenesis
- Treatment
- RAYNAUD’S PHENOMENON
- Definition
- Chronic Interstitial Nephritis
- NORMAL INTESTINAL PHYSIOLOGY
- Factors Involved in the Choice of Type of Dialysis
- SUDDEN CARDIAC DEATH
- PRE-EXCITATIOIi SYNDROMES
- Hepatorenal Syndrome
- Alterations in Drug Doses in Patients with Renal Failure
- Pyuria
- CLINICAL PRESENTATION
- ACID-PEPTIC DISEASE
- Pulmonary Vasculitis
- Studies of Pancreatic Structure and Function
- Etiology and Pathogenesis
- Pneumonia in the Immunocompromised Host
- Pathogenic Mechanisms
- Initial Assessment
- History and Physical Examination
- CHEST WALL DISEASE
- OBLITERATIVE OR OBSTRUCTIVE PULMONARY HYPERTENSION
- Amyloidosis
- CARDIOMYOPATHY
- SMOKE INHALATION
- SPECIFIC CAUSES OF CIRRHOSIS
- Magnetic Resonance Imaging (MRI)
- Radionuclide Imaging
- Other Glomerulonephritides
- CAUSES OF PULMONARY HYPERTENSION