Sodium Retention
Renal salt retention, expressed clinically either as. edema formation or as volume overload and hypertension, is another common feature of glomerular disease. A reduction in GFR may account for some of the decreased ability to excrete salt, but other factors are usually present. Alterations in glomerular hemodynamics may independently compromise the ability of the kidney to excrete sodium. In this case, a change in glomerulotubular balance leads to increased salt and volume absorption in the proximal nephron. A decrease in the effective circulating blood volume due to a reduction in oncotic pressure may stimulate the renin-aldosterone axis and lead to hormone-dependent renal sodium retention in nephrotic patients. Renin and aldosterone secretion may also be increased in cases of nephritis in which renal blood flow is significantly reduced.
- CLINICAL APPROACH TO LIVER DISEASE
- NONMEDICAL MANAGEMENT OF ANGINA PECTORIS
- The Fanconi Syndrome
- Incidence
- Alterations in Drug Doses in Patients with Renal Failure
- Clinical Presentation
- Renal Glycosuria
- Focal Glomerular Sclerosis (FQS)
- BILIRUBIN METABOLISM
- OBLITERATIVE OR OBSTRUCTIVE PULMONARY HYPERTENSION
- Pulmonary Infiltrates with Eosinophilia PIE
- ORIGIN OF ABDOMINAL PAIN
- APPROACH TO THE PATIENT WJTH SUSPECTED MALDIGESTION AND/OR MALABSORPTION
- CONSTRICTIVE PERICARDITIS
- HHSC Legislative Appropriations Request (LAR)
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- Classification or Glomerular Diseases
- TREATMENT OF MALABSORPTION
- CLASSIFICATION AND PATHOPHYSIOLOGY
- Nosocomial Pneumonia
- Women’s Health Program
- Incidence
- COMMON PRESENTING COMPLAINTS
- Hepatic Encephalopathy
- New Eligibility System
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- Upper GI Bleeding
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- HEART DISEASE AND PREGNANCY
- LIMITATION OF MFARCT SIZE
- Beta Blockers